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Unlike conventional chemotherapeutic agents, curcumin can simultaneously modulate several critical oncogenic signaling pathways implicated in prostate cancer pathogenesis, such as phosphoinositide 3-kinase/protein kinase B/mammalian target of rapamycin, nuclear factor kappa B, Wnt/-catenin, signal transducer and activator of transcription 3, and androgen receptor signaling (Shanmugam et al., 2015

Alakkas et al., 2019

Notably, the LY flux patterns in FA iBMVEC closely resemble those of the cell-free blank (CFB), indicating that these patient-derived cells are significantly hyperpermeable and lack the TJ maturation patterns characteristic of barrier-forming cells

The mechanisms of malic enzyme 2 in the tumorigenesis of human gliomas

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monocytogenes (Hedberg, 2011)