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In Gclm KO mice and other models involving a weakened antioxidant capacity, the OxS is more prominent in PV neurons as compared to other types of GABAergic or principal neurons [78,79,80]

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In some cases of pernicious anemia, parietal cell auto-antibodies lead to the destruction of IFproducing parietal cells in the stomach resulting in B12 malabsorption, and thus deficiency

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Furthermore, as mentioned previously, bacteria are far too large and lipophilic to be transported via the paracellular pathway, even in the presence of inflammation [42, 72]

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