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We report PBDE-induced glutamate dysregulation, often associated with the development of ASD, independent of neuroinflammation

Several studies have pointed out that ASD subjects of all ages present nutritional abnormalities and redox imbalances caused by the dysfunctional mitochondria ( Several studies have proposed that ASD may involve different biological profiles, including alterations in redox balance, mitochondrial dysfunction and immune-inflammatory signaling

& Schmeisser, K

It also decreased nitric oxide synthase (NOS) activity, thus modulating the nitric oxide pathway involved in itch genesis

doi: 10.1080/15476278.2016.1145318 16 LamotheSBernardVChristin-MaitreS

The researchers concluded that aged cells consume more NADH and glutathione simultaneously, producing a compounded redox decline that younger cells do not experience