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Specifically, the mechanisms by which CAP achieves pain relief in these contexts are multifaceted and directly address the pathophysiological sources of nociceptive pain
Overexpression of atypical protein kinase C in HeLa cells facilitates macropinocytosis via Src activation
14,15 Transcription factors such as FOXO3 and p53 stimulate autophagy in this context, acting as tumor suppressors

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