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Pharmacological targeting of GSK-3 and NRF2 provides neuroprotection in a preclinical model of tauopathy

Neuroinflammation and the immune system Microglia mediate the immunomodulatory processes in the brain and play a crucial role in the neuroinflammation in PD, leading to chronic activation and releasing of pro-inflammatory cytokines such as tumor necrosis factor-alpha (TNF-) and interleukin-1 beta (IL-1), which further increase the damage in neurons and promote disease progression 41

[1, 2] G6PD deficiency, an X-linked recessive disorder, is the major cause of decreased G6PD activity

Environmental factors such as pollution, ultraviolet radiation, and exposure to mutagenic and carcinogenic agents can damage body cells by generating reactive oxygen species (ROS), which leads to oxidative stress

Induction of HO-1 and redox signaling in endothelial cells by advanced glycation end products: A role for Nrf2 in vascular protection in diabetes

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