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Mechanistically, a key way MOTS-c influences fat metabolism is through its upstream activation of AMPK and downstream effects on gene expression

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In preclinical research the same activity has been linked to angiogenesis (endothelial-cell migration and tubule formation, a different pathway than BPC-157's growth-factor route), to anti-apoptotic, pro-survival signaling , and to progenitor-cell support and matrix stabilization

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