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Underwood R

The apoplast, a critical site for host-pathogen interactions, benefits from extracellular or apoplastic GPXs that reduce ROS levels, thereby helping the plant prevent tissue damage (Gaber et al., 2012)

Ferroptosis is a novel mechanism for ZnONP-induced endothelial cytotoxicity, and NCOA4-mediated ferritinophagy is required for ZnONP-induced ferroptotic cell death (Qin et al

Phylogenetic analysis of the protein-encoding genes of the major redox systems have shown that C

Furthermore, p53 has been shown to regulate cell metabolism through crosstalk with various cellular pathways, such as the mammalian target of rapamycin (mTOR) and AMP-activated protein kinase (AMPK) pathways [72]
