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22, G/D-CuP group exhibited a significantly reduced proportion of CD86-positive cells compared to the NC group

The most important mechanisms of the hypertension are explained by the VEGF blockade inducing endothelial dysfunction resulting in the inhibition of VEGF-dependent vasodilatory pathways such as nitric oxide and prostacyclin, as well as the possible up-regulation of vasoconstrictive pathways such as endothelin-1

For systemic vasculitis, particularly for antineutrophil cytoplasmic antibody-associated vasculitis (AAVs), EULARs management guidelines do not list PN as a part of the life-threatening situations, suggesting therefore the combination of glucocorticoids (1 mg/kg

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