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Although existing studies have elucidated the genetic basis of IMF deposition in chickens from the perspectives of interspecific and intraspecific differences, several limitations remain: 1) Current genetic mechanism studies have predominantly focused on specific genes or pathways (e.g., FABP4 and PPAR), yet they lack systematic integration of multidimensional regulatory networks (e.g., the interactions among SNPs, epigenetic modifications, and noncoding RNAs (ncRNAs))

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TXG protects the colon by reducing the total inflammatory index, infiltration of inflammatory cells, submucosal edema, goblet cell loss, epithelial erosion, granulation tissue, epithelial hyperplasia, and crypt irregularity, abscesses and loss (124, 125)

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