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[2] Exaggerated sympathetic output below the cord lesion: Pale, cool limbs

Notably, inherited mutations in HFE, HJV, and TFR2 lead to hereditary hemochromatosis, a disease of iron excess resulting in dysfunction of multiple organs including liver, pancreas, heart, and endocrine glands

Enhancement of fibroblast growth factor (FGF) activity by an FGF-binding protein

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The predominant mechanism involves Tf/TfR1-mediated endocytosis, where transferrin-bound Fe 3+ binds to TfR1 on endothelial cells, is internalized into endosomes, acidified, and subsequently released into the cytosol via DMT1-mediated transport