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Our key findings include: Only transient glucose lowering, followed by reduction in insulin secretion

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SCFAs have been linked to bloodbrain barrier protection and neuroinflammatory modulation, so interventions that restore SCFA availability could plausibly address the previously described metabolic void driven by functional dysbiosis, rather than acting only as non-specific supplements (

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2, the diffraction measurement results allowed unequivocal tamoxifen identification in the form of citrate (Figure 6B)