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doi: 10.1001/archinternmed.2009.495 MacFarquhar JK, Broussard DL, Melstrom P, et al
The structures of eIF4E-eIF4G complexes reveal an extended interface to regulate translation initiation
Yale J Biol Med
In BALB/c mice, where acute infection is not limited by Ly49H-dependent natural killer (NK) cell responses, cDC1s and cDC2s are crucial to generate protective CD8 T cell responses 25,26,27,28
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Oxidative stress, disturbed calcium homeostasis, and metabolic stress lead to the accumulation of misfolded proteins, triggering UPRER, while ROS and Ca 2+ signaling from damaged mitochondria exacerbate this burden
