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for diabetes as well as certain toxicological and pathological conditions of the liver
J Drug Dev 1988;1(3):163-166
Post-exercise ingestion, especially with a carbohydrate, can aid glycogen resynthesis
When the energy demand is high, inhibition of the isoform ACC2 of acetyl-CoA carboxylase increases fatty acid oxidation, while inhibition of the isoform ACC1 decreases fatty acid synthesis
The blockade of ERK1/2 activation by U0126 (a MAPK kinases inhibitor) attenuated the IRE1 level, suggesting that ERK1/2 activation causes ER stress activation in PFOS-treated RTCs (Fig
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