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These distinct effects of DMF indicate a cellular context-dependent manner in ferroptosis regulation, and more additional studies are required to dissect the mechanisms in erythroblasts

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Wei J, Wang G, Lai M, Zhang Y, Li F, Wang Y, Tan Y

A., Oberye, J

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The clinically normal heterozygous carriers for this disease have about half of the normal glutathione synthetase activity, as predicted, but have normal clinical phenotypes, cellular glutathione levels, and 5-oxoproline excretion (53)