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The rate was calculated over 300 s and expressed as mean SD of at least three independent experiments

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Extrinsic apoptotic pathways are activated by interactions between cell surface exposed death-inducing ligands such as FAS ligands (FASL) and tumor necrosis factor (TNF), and their cognate receptors Fas cell surface death receptor (FAS) and TNF receptor (TNFR) [110, 111]