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Figure 1 Initiated by dysbiosis, harmful gut-derived products including LPS and FFAs translocate into the liver via the portal circulation

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We interpret our finding as the urgent need for microtubule polymerization resulting in fast and deformed growth, which is then repaired upon the normalization of neuronal homeostasis

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Hemin-induced HO-1 was shown to mitigate cytokine storm and tissue injury in murine models of sepsis and renal damage (89), while CoPP-induced HO-1 was shown to inhibit influenza A and RSV viral replication (90)