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In addition, it disrupts oxidative phosphorylationmediated cuproptosis and metabolic reprogramming through mitochondrial proteolipid acylation, thereby impairing insulin and energy metabolism (58, 82, 83) and accelerating the progression of DM and its complications (57, 84)

[PubMed] [Google Scholar] 74.Miller L, Thompson K, Pavlenco C, Mettu VS, Haverkamp H, Skaufel S, et al

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Before billing modifier 51, clinicians should confirm whether NCCI bundling edits allow separate reimbursement