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These verdicts characterize the regulation of sortilin shedding and identify a novel mechanism by which sortilin ectodomain shedding acts as a regulatory switch for delivery of BDNF to the secretory pathway or to the lysosome, thus modulating the bioavailability of endogenous BDNF (Fig

Restoration of NAD + levels by administrating the precursor NR provides greater mitochondrial resistance against impairment of redox balance and, therefore, could play a key role in those diseases that are associated with mitochondrial dysfunction and OS

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