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Collectively, these findings establish ROS as critical enablers of HCC metastasis, orchestrating EMT, proteolytic matrix degradation, and distant organ colonization through coordinated activation of ERK/AKT, JNK, and NF-B signaling [91], thereby providing a mechanistic basis for the link between oxidative stress and poor clinical outcomes in HCC

Astroglia in the vulnerability to and maintenance of stress-mediated neuropathology and depression

10.1186/s40478-019-0696-4 Acta Neuropathol

DoxPt generally displayed a potency either similar to doxorubicin (Fig

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