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In vitro, miR-233 5p had the ability to polarize M1 macrophages towards an M2 phenotype, with increased expression of anti-inflammatory gene Arg-1, and suppression of proinflammatory cytokines TNF-, IL-1, and IL-6 (255)

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Because the absolute numbers of CD11b + Gr-1 low cells were similar between -toxin-treated and untreated conditions, the increase in the CD11b + Gr-1 low cell population in -toxin-treated BMCs reflected the loss of CD11b + Gr-1 high cells (Fig