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The hypothesis that CYP2E1 modulates myocardial pathologies via mitochondrial biological processes was tested through transcriptomic and proteomic analyses using CYP2E1-specific animal models and stable H9c2 cell lines overexpressing Flag-CYP2E1 (Fig

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Noteworthy, [Ca 2+ ] i levels must increase to ~250350 nM to trigger glutamate release from astrocytes [208]