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Our growing understanding of redox-mediated cellular events, however, suggest that some ROS is essential for normal cellular processes, such as disulfide bond formation and growth factor receptor-mediated signaling, which requires oxidative inactivation of certain phosphatases

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The protein expression of the four genes in culture medium were further tested with ELISA kits

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Here, we show that sustained iron overload, but not acute exposure, leads to a state of ferroptotic stress where nerve cells remain viable but become hypersensitive to oxidative injury