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doi: 10.1517/14656566.2012.725045 6 MomoedaMAkiyamaSTanakaKSuzukamoY

We anticipate that providing a novel mechanistic separation for SCLC subtype biology and plasticity may on the one hand benefit patient stratification and on the other hand facilitate informed treatment approaches that take selective cell death pathway availability in SCLC subtype heterogeneity and plasticity into consideration

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Furthermore, numerous studies have shown that in addition to ROS-mediated modification of the Keap1 inhibitor protein, ARE inducers also affect signalling cascades involved in the regulation of Nrf2

Manganese activates NLRP3 inflammasome signaling and propagates exosomal release of ASC in microglial cells

doi: 10.1177/1747493018778713