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Although transcription errors, translation errors, or protein damage are not permanent, such as DNA mutations, due to constant protein turnover by dilution and degradation, they may still accumulate over time

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SOD1-mutant mice exhibited mitochondrial ultrastructural pathologies in spinal cord and skeletal muscle tissues, manifesting as reduced mitochondrial length, cristae disorganization, and increased spherical fragmented mitochondria (Mendez-Lopez et al., 2021)

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Koo S, Park OK, Kim J et al (2022) Enhanced Chemodynamic Therapy by Cu-Fe Peroxide Nanoparticles: Tumor Microenvironment-Mediated Synergistic Fenton Reaction