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Yu et al., 2013)

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on the other hand, it is both a major product of mitochondrial dysfunction and a contributor to further mitochondrial damage [59]

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This screening step revealed an increase in the levels of a hydrogen sulfide (H 2 S) and polysulfides (H 2 S/polysulfides)producing enzyme, Mpst (3mercaptopyruvate sulfurtransferase

In the study of cisplatin-induced apoptosis of renal epithelial cells, Kim et al
