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Evidently, intracellular cyst(e)ine contribute to ferroptosis suppression via at least two additional pathways in different contexts: in a mouse lung adenocarcinoma model, NFS1 cysteine desulfurase utilizes cysteine to synthesize ironsulfur clusters, which are necessary for preventing activation of the iron-starvation response, subsequent iron influx and ferroptosis 14 (Fig

Globus MY-T, Busto R, Dietrich WD et al (1988) Intra-ischemic extracellular release of dopamine and glutamate is associated with striatal vulnerability to ischemia

Impact of 18-month soy protein supplementation on steroid hormones and serum biomarkers of angiogenesis, apoptosis, and the growth hormone/IGF-1 axis: results

In overdose, the increased formation of NAPQI depletes glutathione and once glutathione is depleted by more than two-thirds it covalently binds to critical cellular proteins

To this end, multiple studies reported matrix metalloprotease- and ADAM-dependent cleavage sites at Arg31-Leu32 and Pro52-Leu53 that were inhibited by O-glycosylation at Ser 37 /Ser 41 (Goth et al., 2017