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Promoter-specific activation and demethylation by MBD2/demethylase

In addition to biocompatibility, biodegradation, bioadhesiveness, and biofunctionality of the NDBs, several drawbacks must be addressed, such as low stability, low melting point, high surface tension, structural complexity, and well-known immunological response from organisms (Ige et al., 2012

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Integrative proteomics, genomics, and translational immunology approaches reveal mutated forms of Proteolipid Protein 1 (PLP1) and mutant-specific immune response in multiple sclerosis

(2003) investigated Epitalon (the parent compound of NA Epitalon) in senescent fibroblast cultures and observed reactivation of telomerase activity and extended replicative lifespan, providing the mechanistic basis for the stabilized derivative [1]

PLoS One 12, e0173402