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Ma G, Zhang Z, Li P, Zhang Z, Zeng M, Liang Z, Li D, Wang L, Chen Y, Liang Y, Niu H

The most primary and urgent aspect is to provide authoritative definitions and interpretations of doseresponse models for hormesis from a toxicological perspective

FIGURE 2 2.2 Blocking the cascade reaction of MAPK signaling pathway MAPK pathway regulates the expression of pro-inflammatory genes through ERK, JNK and p38 three branches ( Zeyen et al., 2022

& Thorson, J

Intriguingly, however, Cu (II)-atsm does not improve astrocyte dysfunction by increasing SOD1 activity but by decreasing mitochondrial respiration, which is increased in these cells, and by increasing glycolysis (Dennys et al., 2023)

The mechanism of lipid peroxidation begins with the attack of oxidants, such as free radicals, targeting the carbon-carbon double bonds within the PUFAs