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Endothelial deficiency of insulin-like growth factor-1 receptor (IGF1R) impairs neurovascular coupling responses in mice, mimicking aspects of the brain aging phenotype

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In cirrhosis the failure to properly replace destroyed hepatocytes and the excessive collagen deposition to distort blood flow through the liver (portal hypertension) result in severe liver dysfunction

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Activated Akt then phosphorylates eNOS at Ser1177, directly increasing nitric oxide production