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ATP7A-LINKED NEURODEGENERATIVE DISORDERS Unlike AT7B, where mutations in the gene result in a single known clinical phenotype (Wilson disease), mutations in ATP7A cause three X-linked neurodegenerative diseasesMenkes disease, occipital horn syndrome (OHS), and X-linked distal hereditary neuropathy (dHMN) (Kaler, 2011)

30 In addition, the downregulation of NDUFS1, another complex I subunit, is selected by antiangiogenic therapy in ovarian cancer, leading to a stable glycolytic phenotype and increased aggressiveness

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Research has shown that oxidative stress plays a significant role in COVID-19 pathology, and antioxidant therapy may help mitigate this damage