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Mechanistically, the SAM derived from one carbon metabolism increases the promoter abundance of H3K27me3 of insulin-like growth factor 1 (IGF1), which in turn results in the inactivation of p38-dependent JAK-STAT1 axis and promotes the STAT6-mediated M2 polarization (69)

[DOI] [PubMed] [Google Scholar] Ne G., Zaffagnini M., Trost P., Issakldis-Bourguet E

FIGURE 10 10 Metal chelators Various chelating agents have been used to inhibit metal-dependent oxidative damage

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