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Srkzy, M
Inhibiting IDH1 reduces damage and apoptosis in Ox-LDL-treated macrophages while increasing Nrf2 levels, thereby decreasing foam cell formation (Li B
doi: 10.1007/s00109-011-0845-0
3C and Supplementary Data S3)
Since the cellular synthesis of glutathione is ATP-dependent, decreased ETC activity also undermines glutathione synthesis, further exacerbating oxidative stress in a vicious cycle [8]
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