glutathione for schizophrenia deficiency in the early postnatal developmental period as a neurodevelopmental animal model of | Pharmacological Reports Factors that contribute to the
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Herein, using conditional Rictor knockout mice, we were able to demonstrate that Rictor deletion disrupted glutathione metabolism through AKT/Nrf2 signaling pathway and induced intracellular oxidative stress during the malignant transformation of Kras/Pten-mutant ovarian surface epithelial cells
