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doi: 10.3389/fphar.2021.685898

Based on the increased oxidative stress following MDMA exposure, we hypothesized that oxidative modifications of certain mitochondrial proteins lead to inhibition of their activities, resulting in mitochondrial dysfunction and ultimately contributing to MDMA-mediated organ damage

This work was supported by the National Key Research and Development Program of China (grant 2018YFC2001802 to X

PM(2) (.5) exposure-induced ferroptosis in neuronal cells via inhibiting ERK/CREB pathway

Crucially, ischemia injury impairs the integrity of the intestinal epithelial barrier, resulting to increased permeability