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However, it was observed that instead of resulting in increased intracellular EGT levels, it instead replenished EGT as it was dissipated in the process of detoxifying H 2 O 2 , consistent with data from Quantitative proteomic analysis demonstrated differential abundance of a multitude of reductases, oxidases, stress response proteins and enzymes with oxidizing products in egtA 26933 compared to A

Helicobacter pylori inhibits autophagic flux and promotes its intracellular survival and colonization by down-regulating SIRT1

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Fungal cells penetrate the BBB either through transcytosis across endothelial cells lining cerebral vessels or via a Trojan horse mechanism involving carriage within phagocytes, ultimately leading to life-threatening meningoencephalitis (Kronstad et al., 2011)