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Long-chain n-3 fatty acids attenuate oncogenic KRas-driven proliferation by altering plasma membrane nanoscale proteolipid composition

They can regulate lipid metabolism by directly activating target genes such as CPT1A

Niaz, E

The assays were performed using the compound concentration range of 31.25 nM3 M in 1 PBS-P+ buffer and 2% DMSO as running buffer

TBARS Thiobarbituric acid reactive substances
