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It is assumed that after the initial driver mutation, a senescence program stops the growth of MN
Heme oxygenase-1 regulates cardiac mitochondrial biogenesis via Nrf2-mediated transcriptional control of nuclear respiratory factor-1
HDL Function and Atherosclerosis: Reactive Dicarbonyls as Promising Targets of Therapy
[DOI] [PMC free article] [PubMed] [Google Scholar] 183
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