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Therefore, it is not surprising that mutations in this enzymatic machinery contribute to cancer development [149]

Proc Natl Acad Sci USA (2021) 118(5):e2009156118

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The main reasons for this are twofold: First, although the rectum and parts of the distal colon in humans have no direct vagus nerve innervation, VNS can activate the vagus afferent fibers, project to the central solitary nucleus (NTS), thereby enhancing the output activity of the sacral nerves and regulating the functions of the distal colon and rectum, achieving the therapeutic effect (31)

Similarly, the deletion of TFRC prevented ferroptosis induced by erastin or cystine deficiency [26, 27]

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