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Recently a meta-analysis examined the possible link between mitochondria and autism, the conclusions of this study are that it is not clear if mitochondrial dysfunction contributes to the development or pathogenesis of ASD or if mitochondrial dysfunction is just an epiphenomenon of ASD[14]

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The short half-life reflects rapid clearance and ligand exchange with serum proteins such as albumin and ceruloplasmin

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doi: 10.1105/tpc.104.027920 60 MorohashiK.ZhaoM.YangM.ReadB.LloydA.LambR.et al

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& Renard, J

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