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BH4 acts both as a direct trap for small molecule free radicals and a cofactor for enzymes involved in CoQ10 synthesis, protecting phospholipids containing two polyunsaturated fatty acid (PUFA) tails from oxidative degradation during ferroptosis

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Such formulations have demonstrated improved half-life and sustained release, making them viable for combination with conventional therapies

Moreover, it was found that besides NAD(P)H production, 1C flux also maintains redox homeostasis via GSH production [80]

161 Other researchers have also shown that increasing the levels of ROS through lipid peroxidation of biological membranes is a promising strategy for inducing ferroptosis

Both formulations are available following a medical consultation