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doi: 10.3389/fimmu.2022.975921 32 BenakisCBreaDCaballeroSFaracoGMooreJMurphyMet al

Clinical studies of ASD and experiments on animal and cellular models of this disorder have revealed the biochemical endophenotype of insufficient mitochondrial energy production

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n=141 ALC, n=139 control), the pooled estimate also favored ALC, though the confidence interval included the possibility of no effect (SMD 1.33, 95% CI 2.80 to 0.14

1a trat eine signifikante Reduktion auf, whrend gemss Asadi et al