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Description
10.1016/j.ajo.2024.08.030
The pathophysiology of MASLD is complex and multifactorial, involving hepatic lipid accumulation driven by excess adiposity, systemic inflammation, insulin resistance, and activation of hepatic stellate cells (HSC)-factors that collectively contribute to liver injury and fibrogenesis
effects probably related to its anti-diabetic and weight-lowering properties
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The abdomen is an FDA-approved injection site for these medications, offering accessibility and consistent subcutaneous tissue
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