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The present results indicated that the antihypertensive effect of 1 AR-selective antagonists may be explained by their ability to hamper norepinephrine release through presynaptic modulation, in agreement with previous analyses showing that the hypotensive effect of AR antagonists, including atenolol, did not depend on cardio depression or suppression of renin secretion (31)
D., Koch, H
GCaMP3/glr3.3 and GCaMP3/glr3.6 mutants were generated by CRISPR/Cas9 in WT plants overexpressing GCaMP3
(2020) 11:826doi: 10.3389/fneur.2020.00826 [DOI] [PMC free article] [PubMed] [Google Scholar] 60
Promotion of cancer cell migration: an insulin-like growth factor (IGF)-independent action of IGF-binding protein-6
