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Removal of the first 14 residues of GLP-1 yielded a peptide with 1500-fold lower affinity than GLP-1 and with antagonist properties, while removal of the C-terminal 11 residues resulted in undetectable binding and activity (Gallwitz et al ., 1990)

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As sDPP4 is an adipokine upregulated in obesity and T2DM that triggers IR and metabolic complications (4, 5), it might be speculated that the beneficial effects of DPP4 inhibitors would be higher in those early phases of the metabolic disorders previous to the development of established cardiovascular disease

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