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Given the increasing evidence of alternative receptor-independent sources of cAMP (from intracellular sAC) (29, 30), we hypothesized that GLP-1(2836) mediates its GLP-1Rindependent effects through sAC

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The impact of their molecular mechanisms has been evidenced by landmark trials, which have repeatedly demonstrated that certain GLP-1 RAsincluding liraglutide, semaglutide, and dulaglutideare particularly efficacious in reducing MACE, especially in patients with established ASCVD