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Tirzepatide slows gastric emptying, which could exacerbate symptoms

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Outcomes Outcomes were assessed at 1, 5, and 8 years following the index date and included: Primary outcomes: all-cause mortality and overall survival Liver-related outcomes: advanced liver disease (ALD) was defined according to clinical diagnoses (e.g., portal hypertension, ascites, varices, hepatic encephalopathy), laboratory abnormalities (e.g., thrombocytopenia, hyperbilirubinemia, hypoalbuminemia, hyperammonemia), or dispensed medications used specifically for cirrhosis (e.g., propranolol, lactulose, rifaximin, spironolactone)

[DOI] [PMC free article] [PubMed] [Google Scholar] 171.Cant C., Houtkooper R.H., Pirinen E., Youn D.Y., Oosterveer M.H., Cen Y., Fernandez-Marcos P.J., Yamamoto H., Andreux P.A., Cettour-Rose P., et al

acting upon different organs such as the pancreatic islets, the hypothalamus or the stomach, for the release of insulin, to regulate food intake or gastric emptying respectively ( Surprisingly, this heterogeneous and highly plastic population of cells is known to differentiate from a single staminal progenitor that gives also rise to enterocytes, goblet and paneth cells ( It has been known for more than a century that the gut is capable to stimulate the endocrine portion of the pancreas and even improve the hyperglycaemic state of diabetic patients ( In the last three decades, the incretin-effect has been attributed primarily to two peptide hormones, the gastric-insulinotropic peptide (GIP) and glucagon-like peptide-1 (GLP-1), excreted primarily by duodenal (K) and ileo-colonic (L) enteroendocrine cells respectively ( Obesity and Type 2 diabetes are chronic diseases for which the most effective treatment is bariatric surgery

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