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To investigate the physiological functions of endogenous GLP-1 mediated via vagal sensory neurons, a rat model with vagal sensory neuron-specific GLP-1R knockdown was generated by microinjecting a viral vector expressing GLP-1R shRNA into the nodose ganglion [51]

Consistently, both treatments also lead to an upregulation of the PPAR-regulated gene Glut4, a protein implicated in glucose uptake in response to insulin (fig

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doi: 10.1186/s13098-024-01412-x

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Importantly, current therapeutic approaches focus on alleviating immune-mediated injury to the kidney, mainly targeting the migratory and functional features of immune cells with little impact on fibrosis or chronicity