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The bulky A350 6.39b W and K351 6.40b A mutants almost abolished the maximal response (Emax) of GLP-1R-mediated cAMP accumulation in presence of compound 2, while V332 5.62b A, K346 6.35b A and L349 6.38b A mutants greatly elevated basal cAMP activities but significantly diminished the efficacy of the response upon stimulation by either compound 2 or GLP-1, suggesting these mutations affect the kink of TM6 required for receptor activation

Additionally, it has been shown to reduce the risk of cardiovascular disease and other health complications associated with obesity

[DOI] [PMC free article] [PubMed] [Google Scholar] 142.Thiele T.E., Van Dijk G., Campfield L.A., Smith F.J., Burn P., Woods S.C., Bernstein J.L., Seeley R.J

All statistical analyses were based on the full analysis set using in-trial data

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