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The initial mechanical disruption of white matter tracts is rapidly exacerbated by multiple secondary sequelae that include ischemia, inward calcium flux, infiltration with inflammatory cells and liberation of high levels of ROS which evolve over time to produce a chronic state of inflammation
Feng S, Wang H, Liu J, Aa J, Zhou F, Wang G
AhR plays a context-dependent role in modulating ferroptosis sensitivity across tumor cell lines (18,19)
As a multi-target therapeutic agent bridging antioxidant defense and anti-inflammatory intervention, SAC provides a safe and effective adjuvant therapeutic option for UC management (Fig
Glutaredoxin exerts an antiapoptotic effect by regulating the redox state of akt
98 Wong J, Magun BE, Wood LJ
