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Other studies have revealed compositional and functional changes in gut microbiota (dysbiosis) and subclinical intestinal inflammation as possible drivers of TJ destabilization and leaky gut in animal models of arthritis and humans with RA [2, 3]

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In phase II trials focusing on dermal applications, TB-500 has displayed promising outcomes for patients with pressure ulcers, stasis ulcers, and epidermolysis bullosa

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SL0722Ra) and IL-6 (Cat

Zubcevic J, Richards EM, Yang T, Kim S, Sumners C, Pepine CJ
